By Alberto Zanchetti (auth.), Michel Emile Safar M.D., Michael Stimpel M.D., Alberto Zanchetti M.D. (eds.)
On the get together of the symposium on high blood pressure in Postmenopausal ladies (Monte Carlo, January 19-22, 1994), foreign scientists and clinicians mentioned some of the elements of hypertension in older ladies. This quantity offers an outline of this significant point of either cardiology and gynecology. specified emphasis is given to different techniques to antihypertension treatment.
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Additional resources for Hypertension in Postmenopausal Women
These larger doses generally lead to an 42 Rogerio A. Lobo increased amount of withdrawal bleeding. Prevention of hyperplasia necessitates a reduction in mitotic activity. This, and a reduction in bleeding, should be the principal aim of an effective regimen. With this approach, it is estimated that the maximal inhibitory effect on cardiovascular function (including arterial as well as lipoprotein effects) would be a 15 % reduction in the benefit for the 10 days of exposure . This is not likely to affect RR estimates of the cardioprotective effects of hormonal replacement.
However, in a short-term longitudinal study , it was observed that after natural menopause, HDL-C did decrease with time. This is also in keeping with studies showing a decrease in HDL-C after the abrupt cessation of ovarian functions such as with oophorectomy or in some studies after the use of a gonadotropin-releasing hormone analog . Much significance has been placed on changes in HDL-C, specifically on the benefits achieved by raising HDL-C with estrogen "replacement" therapy in postmenopausal women.
1992)  Although detailed human studies are lacking, recent work has suggested that estrogen mediates nitric oxide relase as well as having calcium antagonist activity. In women with chest pain and normal coronary arteries (syndrome X), the hyperemic response to occlusion in the forearm was restored after estrogen  (Fig. 6). Recently, sublingual estradiol was shown to improve exercise time and time to ST suppression in women with angina  (Fig. 7). The rapidity of some of these responses has led to the theory that estrogen works in a nongenomic way, possibly by antagonizing calcium channels.
Hypertension in Postmenopausal Women by Alberto Zanchetti (auth.), Michel Emile Safar M.D., Michael Stimpel M.D., Alberto Zanchetti M.D. (eds.)