By American Academy of Ophthalmology, Ramana S. Moorthy MD
Starts with an summary of immune-mediated eye disorder, summarizing uncomplicated immunologic strategies, ocular immune responses and precise subject matters in ocular immunology. Discusses the scientific method of uveitis and studies noninfectious (autoimmune) and infectious types of uveitis, with an increased part on viral uveitis and new fabric on infectious and noninfectious scleritis.
Enhanced detection of infectious brokers by means of immunologic and genetic equipment and biologic therapeutics are exact. additionally covers endophthalmitis, masquerade syndromes, issues of uveitis and ocular facets of AIDS.
Upon of completion of part nine, readers might be capable to:
Identify basic and particular pathophysiologic procedures that impact the constitution and serve as of the uvea, lens, intraocular cavities, retina, and different tissues in acute and persistent intraocular inflammation
Differentiate and establish infectious and noninfectious uveitic entities
Choose acceptable exam recommendations and correct ancillary stories in keeping with even if an infectious or noninfectious reason is suspected
Read or Download 2014-2015 Basic and Clinical Science Course (BCSC): Section 9: Intraocular Inflammation and Uvetis PDF
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Additional resources for 2014-2015 Basic and Clinical Science Course (BCSC): Section 9: Intraocular Inflammation and Uvetis
Subsequently, other factors, such as platelet-activating factor (PAF), various cytokines, and bacterial products, can induce the up-regulation of the ~-integrin family. As integrins are expressed, the selectins are shed, and neutrophils then bind firmly to endothelial cells through the immunoglobulin superfamily molecules. Subsequent to adhesion, various chemotactic factors are required to induce transmigration of neutrophils across the endothelial barrier and extracellular matrix into the tissue site.
The initial steps of the afferent phase of this secondary response are identical to those of the initial exposure. However, if a memory T lymphocyte is present at the cutaneous site, then the processing and effector phases occur within 24 hours at the site, as the memory T lymphocyte becomes activated directly upon interacting with the LC. In addition, some LCs leave the skin, enter the draining node, and encounter memory T lymphocytes there. Processing during the secondary response is much more rapid, and within 24 hours restimulated memory cells enter the circulation and migrate to the toxin-exposed cutaneous site.
On exposure to antigen and antigen-presenting cells (APCs) within the lymph node, the 3 major lymphocyte subsets-B lymphocytes, CD4 T lymphocytes, and CD8 T lymphocytes-are activated to release specific cytokines and perform specific functional activities. B lymphocytes are stimulated to produce one of the various antibody isotypes, whose functions include complement activation, antibody-dependent cellular cytotoxicity (ADCC), agglutinization, allergen recognition, and release into secretions. CD4 T lymphocytes become activated into T helper-1 (Th1) or T helper-2 (Th2) subsets.
2014-2015 Basic and Clinical Science Course (BCSC): Section 9: Intraocular Inflammation and Uvetis by American Academy of Ophthalmology, Ramana S. Moorthy MD